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. 2018 Jan 2;217(1):211–230. doi: 10.1083/jcb.201704071

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© 2018 Izadi et al.

This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms/). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 4.0 International license, as described at https://creativecommons.org/licenses/by-nc-sa/4.0/).

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Figure 4. — Cobl-like is critical for dendritic arbor formation. (A–C) Morphology of Cobl-like–depleted neurons (RNAi#1; B) in comparison to control neurons (scrambled RNAi sequence; Scr.; A) and to neurons coexpressing an RNAi#1-insensitive Cobl-like mutant (Cobl-like*; rescue; C). Bars, 10 µm. (D–F) Quantitative determinations of specific defects in dendritic arborization caused by Cobl-like addressing dendritic branch points (D), dendritic terminal points (E), the summarized length of the dendritic arbor (F), and Sholl intersections (G). Data are mean ± SEM, one-way ANOVA Tukey’s post test (D–F) and two-way ANOVA plus Bonferroni’s post test (G). *, P < 0.05; **, P < 0.01; ***, P < 0.001.