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. 2017 Dec 27;5:125–133. doi: 10.1016/j.toxrep.2017.12.019

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© 2018 The Authors

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

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Fig. 2 — Histopathological changes of imidacloprid toxication (96 h). A) Normal histology of the gill structure (arrows). Control group. H&E. 20 μm. B) Hyperplasia of gill epithelium. Low dose toxic group, gill tissue. H&E. 20 μm. C) Hyperplasia of gill epithelium (arrow) and mucous cell hyperplasia (arrow head). High dose toxic group, gill tissue. H&E. 20 μm. D) Normal histology of liver, vena centralis (arrow). Control group. H&E. 20 μm. E). Hydropic and vacuolar degeneration in hepatocytes (arrow). Low dose toxic group, liver tissue. H&E. 20 μm. F). Diffus hydropic degeneration and focal necrosis (arrow head) in hepatocytes. High dose toxic group, liver tissue. H&E. 20 μm. G) Normal histology of neurons (arrows) in optic lobe. Control group. H&E. 20 μm. H). Normal histology of neurons (arrows) in optic lobe. Low dose toxic group. H&E. 20 μm. I), Degenerated neurons (arrows) in optic lobe. High dose toxic group H&E. 20 μm.