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. 2018 Jan 4;15:1. doi: 10.1186/s12974-017-1027-y

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — The effect of Aβ administration on Cdk5 expression and immunoreactivity in hippocampus. Aβ (0.5 nmol) was injected intracerebroventricularly. a Gene expression for Cdk5 was analysed 3 h and 1, 3, 7 and 14 days after injection by quantitative RT-PCR. b Immunoreactivity of Cdk5 was analysed 3 h and 1, 3, 7 and 14 days after injection by SDS-PAGE and Western blotting. Representative pictures were shown. Results of densitometric analysis were normalised to immunoreactivity of GAPDH, as a loading control. Results are presented as the mean ± SEM from four independent experiments (n = 4)