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. 2017 Oct 3;14(6):7241–7249. doi: 10.3892/ol.2017.7129

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Copyright: © Cheng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Effects of p38 expression on the regulation of tumor cell sensitivity to thalidomide treatment. MTT assay for the survival rate of (A) Eca-109 and (B) TE-1 cells. (C) Flow cytometric apoptosis assay of Eca-109 and TE-1 cells. (D) Western blotting for Ku80 expression following treatment with or without thalidomide in cells with different p38 expression levels. (E) p38 expression in esophageal cancer cells is positively associated with IDO expression. Ku80, Ku autoantigen 80 kDa; p, phosphorylated; IDO, indoleamine 2,3-dioxygenase; FITC, fluorescein isothiocyanate; PI, propidium iodide.