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Thioridazine induced autophagy via the AKT/ERK signaling pathway. Representative western blotting demonstrating the expression levels of P-AKT/AKT, P-ERK/ERK, P-P38/P38 following treatment with of 15 µM thioridazine for various lengths of time (0, 6, 12 and 24 h). GAPDH was used as an internal control. AKT, protein kinase B; ERK, extracellular signal-related kinase; P, phosphorylated.
