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. 2017 Oct 20;191(2):180–188. doi: 10.1111/cei.13058

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© 2017 British Society for Immunology

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Innate lymphoid cell (ILC) cells are persistently depleted in anti‐neutrophil cytoplasm autoantibody (ANCA)‐associated vasculitis (AAV) patients and disease controls. (a–d) Frequencies of ILC subsets in healthy controls (HC), disease controls (DC) and AAV patients in active (AP) and remission phase (RP). AAV subtypes are denoted by circle colour as granulomatosis with polyangiitis (GPA) (), microscopic polyangiitis (MPA) (○) and eosinophilic granulomatosis with polyangiitis (EGPA) (). (e–h) Absolute cell numbers of ILC subsets in DC and AAV patients in active (AP) and remission phase (RP). Data represent median and interquartile range. One‐way analysis of variance (anova) was carried out using the non‐parametric Kruskal–Wallis test and Dunn's multiple comparison post‐test. ***P < 0·001; **P < 0·01; *P < 0·05.

Inline graphic — Innate lymphoid cell (ILC) cells are persistently depleted in anti‐neutrophil cytoplasm autoantibody (ANCA)‐associated vasculitis (AAV) patients and disease controls. (a–d) Frequencies of ILC subsets in healthy controls (HC), disease controls (DC) and AAV patients in active (AP) and remission phase (RP). AAV subtypes are denoted by circle colour as granulomatosis with polyangiitis (GPA) (), microscopic polyangiitis (MPA) (○) and eosinophilic granulomatosis with polyangiitis (EGPA) (). (e–h) Absolute cell numbers of ILC subsets in DC and AAV patients in active (AP) and remission phase (RP). Data represent median and interquartile range. One‐way analysis of variance (anova) was carried out using the non‐parametric Kruskal–Wallis test and Dunn's multiple comparison post‐test. ***P < 0·001; **P < 0·01; *P < 0·05.