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. Author manuscript; available in PMC: 2019 Jan 1.
Published in final edited form as: Microcirculation. 2018 Jan;25(1):10.1111/micc.12423. doi: 10.1111/micc.12423

Figure 2. PKA is required for vasoconstriction of cerebral parenchymal arteries in response to elevated glucose.

Figure 2

A) Representative diameter recordings and B) summary hg-induced constriction in the absence or presence of the PKA inhibitor rpcAMPs (10 μM). A solution containing 0 mM extracellular Ca2+ and the LTCC blocker nifedipine (1 μM) was used to obtain the passive diameter. *P < 0.05.