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. 2017 Dec 1;8(67):111470–111481. doi: 10.18632/oncotarget.22838

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Copyright: © 2017 Li et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A) Expression of selected genes changed after knockdown of CCT3 in MGC-803 cells. qPCR showed upregulation of IRAK2, PRKAA2, CDK2, mTOR, IGFBP3, MET, CDK6 and downregulation of MAP3K7, CCND3 in CCT3 knockdown cancer cells. (t test, P<0.01 for all genes). (B) Western blotting showed increased levels of MAP3K7, CDC42, CCDN3 and decreased levels of CDK2 and CDK6 in the MGC-803 cells after CCT3 knockdown.

(A) Expression of selected genes changed after knockdown of CCT3 in MGC-803 cells. qPCR showed upregulation of IRAK2, PRKAA2, CDK2, mTOR, IGFBP3, MET, CDK6 and downregulation of MAP3K7, CCND3 in CCT3 knockdown cancer cells. (t test, P<0.01 for all genes). (B) Western blotting showed increased levels of MAP3K7, CDC42, CCDN3 and decreased levels of CDK2 and CDK6 in the MGC-803 cells after CCT3 knockdown.