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Network analysis depicting menin in complex with JunD, Sin3A, Sap30, and RBBP4 mediating histone deacetylase 1/2 (HDAC1/2)-dependent histone deacetylation and suppression of angiogenesis (VEGF-A) and inflammation (IL-6). Jun–TNF-α drives survival in a HDAC1/2-independent manner by antagonizing menin–JunD–HDAC1/2 activity. QIAGEN’s Ingenuity® Pathway Analysis (IPA®; www.qiagen.com/ingenuity; QIAGEN, Redwood City, CA, USA).
