Table 1.

Spacer Sequences^a

spacer no.	gene	sequence (5′ to 3′)	C (%)	G (%)	CG (%)
1	23–2	aagaacttccaaccggtaat	25	15	40
2	20–1070	gcaatatggaagatattcgt	10	25	35
3	Mrh.2–24	ataatatctaaatcttcatt	15	0	15
4	39.1–151	atgttctgggctgttcttta	15	25	40
5	dda.1–130	gagcagtatcgattagcttt	15	25	40
6	segF496	gatttcagaaggaacttcaa	15	20	35
7	rnlb270	gttgtatcttatcaagtctt	15	15	30
8	srh-8	ttctgaagaagatgcttgaa	10	25	35
9	dexA523	cttccaaagggaactttaga	20	20	40
10	cef154	tcacgaaatacaccataaat	25	5	30
11	39–37	gaacatatcaaaaagcgtag	15	20	35
12	24.2–181	ttacagaagaaattggtgat	5	25	30
13	20–995	gcgctgcaaccaatagtctt	30	20	50
14	39–96	gcgctttatgtttggtaaat	10	25	35
15	24–38	gattgagttgctattcgttg	10	30	40
16	e148	gttagtaaatcctgccacac	30	15	45
17	segD36	tattcgcaattggataaatg	10	20	30
18	uvsy263	agcggataaggatgttttaa	5	30	35
19	23–1490	gaatagaaggcataccgctc	25	25	50
20	uvsy287	tgatacctcgttgcagtatt	20	20	40
21	56–136	aatttcgagctggtcttcgg	20	30	50
22	56–63	tttcttccgcattcattcca	35	5	40
23	e44	caacgtttagaactggcact	25	20	45
24	IP3–28	ggcctttactacagaagctt	25	20	45
25	IP3–10	accacgggctgcattagcaa	30	25	55

^aSequences of the spacers used in the current study. Five spacers (spacers 1–5), which showed similar EOP for both WT and T4(C) mutant infections, are highlighted in bold font.