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. Author manuscript; available in PMC: 2019 Feb 1.
Published in final edited form as: Clin Endocrinol (Oxf). 2017 Dec 14;88(2):243–250. doi: 10.1111/cen.13522

Figure 1.

Figure 1

Total proportion of variance in each outcome explained by genetic (A) and environmental (C or E) factors. Estimates for testosterone and estradiol are from a bivariate model that allowed for quantitative sex differences in all paths and qualitative sex differences in testosterone. Pubertal status results are from a univariate model that allowed for quantitative and qualitative sex differences. Female-restricted participants were off hormonal contraceptives, and in the follicular phase of their menstrual cycle.