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. 2018 Jan 17;9:263. doi: 10.1038/s41467-017-02499-9

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Magnetic resonance imaging of fetal brains. A T2W brain template generated from 16 control fetuses at gestation age 135dGA ± 2d (a), a typical control at 135dGA (b), and the 5 ZIKV-exposed fetal brains (c) are shown in sagittal (left) and axial (right) views at the indicated slice locations (insets). Anterior/posterior, superior/inferior, lateral/medial, and left/right are marked on the 3D brain surfaces (insets) and template images (a). Despite the absence of microcephaly, a thinner somatosensory gyrus (c, red arrows), and a missing secondary sulcus (a–c, yellow arrows), was identified for animal F35467, but not other ZIKV-infected animals