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. 2017 Nov 17;15(1):908–916. doi: 10.3892/ol.2017.7434

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Copyright: © Sun et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — XN inhibited Notch1 activity. (A) XN inhibited Notch1-dependent luciferase activity. Luciferase data were normalized to β-galactosidase and are presented as the means ± SD. *P<0.05, **P<0.01 vs. control. (B) Notch 1 and Hes1 protein expression levels were detected by western blotting. (C) XN inhibited Notch1-specific downstream gene Hes1 and Hey1 mRNA expression levels. DLL-4; anti-delta-like 4; DAPT, Duration of Dual Antiplatelet Therapy; Ctrl, control; XN, xanthohumol; Hes1, hairy enhancer of split 1; hey1, Hes related family BHLH transcription factor with YRPW motif 1.