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. 2017 Nov 9;15(1):991–997. doi: 10.3892/ol.2017.7381

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Copyright: © Yao et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — miR-544 promotes colorectal cancer progression. (A) Expression levels of miR-544 in HT29 cells transfected with miR-544 mimic determined using RT-qPCR. (B) MTT analysis of miR-544-overexpressed HT29 and control cells. (C) Colony formation assay of miR-544-overexpressed HT29 and control cells. (D) Transwell assay of miR-544-overexpressed HT29 and control cells (magnification, ×400). (E) Expression levels of miR-544 in HCT116 cells transfected with miR-544 inhibitor determined using RT-qPCR. (F) MTT analysis of miR-544-depleted HCT116 and control cells. (G) Colony formation assay of miR-544-depleted HCT116 and control cells. (H) Transwell assay of miR-544-depleted HCT116 and control cells (magnification, ×400). **P<0.01, ***P<0.001 vs. control. miR, microRNA; RT-qPCR, reverse transcription-quantitative polymerase chain reaction.