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. 2017 Jul 5;92(1):225–240. doi: 10.1007/s00204-017-2022-x

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© The Author(s) 2017

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 8 — AHRR knockdown increases XRCC6 mRNA levels. a AHRR knockdown increases XRCC6 mRNA levels in MCF7 cells treated for 24 h with DMSO (D) or TCDD (T). b No significant changes in DESI1 mRNA levels were observed after AHRR knockdown in MCF7 cells treated with DMSO (D) or TCDD (T). RNAi-mediated knockdown of AHRR effectively reduced TCDD-dependent increases in AHRR protein levels. Data shown are representative of three independent experiments. Asterisks denoted significantly different (P < 0.05) gene expression differences compared with non-targeted (NT) DMSO-treated cells