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. 2018 Jan 19;8:1187. doi: 10.1038/s41598-018-19459-y

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© The Author(s) 2018, corrected publication 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Hypophosphorylation of rpS6 in IE brain. (a) Example of rp S6 labeling (Ser235/236 and Ser 240/241) from fields taken in the layers V/VI of the visual cortex of P12 SC and IE rats. Calibration bar: 100 µm. (b) Quantitative analysis of rp S6 immunofluorescence intensity in the visual cortex of P12 animals. SC animals showed higher rpS6 expression in comparison to IE animals both for the site Ser235/236 and for the site Ser240/244 (SC P12, n = 5; IE P12, n = 5, Two way ANOVA, post-hoc Holm-Sidak method, p < 0.05). (c) At P21 rp S6 expression did not differ between SC and IE animals (SC P21, n = 5; IE P21, n = 6; p = 0.48). Histograms represent average values ± SEM. *p < 0.05.