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. 2018 Jan 19;8:1250. doi: 10.1038/s41598-018-19638-x

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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EBOV infection of the salivary glands and thyroid. (a) Necrotizing adenitis of parotid salivary gland. Focal acinar cell necrosis with rare large eosinophilic to amphiphilic intracytoplasmic inclusion bodies (arrows, H&E). (b) Infiltrating macrophages, interacinar fibroblasts, and viable and degenerate acinar cells (EBOV VP40 IHC: DAB chromogen and hematoxylin). (c) Infected acinar cells, macrophages and fibroblasts (EBOV NP ISH: fast red chromogen and hematoxylin). (d) Degeneration, sloughing, and necrosis of follicular epithelium, with rare large eosinophilic to amphiphilic intracytoplasmic inclusion bodies (arrows) in follicular cells and infiltrating macrophages (H&E). (e) Infected follicular cells, intra-follicular and interstitial macrophages, and interstitial stromal cells (EBOV VP40 IHC: DAB chromogen and hematoxylin). (f) Infected follicular epithelium and interstitial cells (EBOV NP ISH: fast red chromogen and hematoxylin).