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. 2017 Nov 30;46(2):582–592. doi: 10.1093/nar/gkx1165

Figure 5.

Figure 5.

Alternative splicing is infrequent among coding exons. (A) The percentage of exonic regions (Y-axis) is shown for three subsets of exons: (i) exonic regions with TDU due to alternative splicing [DEU (AS)], (ii) exonic regions with TDU without evidence of alternative splicing [DEU (NAS)] and (iii) a background set of exons matched for expression and exon width. Each color represents a different category of exons according to transcript biotypes: exons coding for principal transcript isoforms [Coding (PI)], exons coding for non-principal transcript isoforms [Coding (non-PI)], 5′ UTRs, 3′ UTRs and exons from non-coding processed transcripts [Processed transcripts]. (B) Sashimi plot representation of the RNA-seq data from frontal cortex and cerebellum of individual WL46. The lower data track shows the transcript isoforms of the gene PKD1. The transcripts are colored according to their biotype (the color legend is the same as in Figure 5A). The highlighted exon (E051) belongs to a non-coding transcript and is differentially spliced across tissues. (C) Same as in Figure 5B, but showing data from tibial artery and whole blood of the individual ZTPG. Transcripts from the gene MAN2B2 along chromosome 4 are shown. The highlighted exon (E018) belongs to a non-coding transcript and is differentially spliced across tissues. (D) Same as in Figure 5B, but showing data from esophagus tissue (muscularis) and heart tissue (left ventricle) of the individual 111YS. The lower track shows the transcripts annotated for gene NISCH along chromosome 3. The highlighted exon (E009) belongs to a non-coding transcript and is differentially spliced across tissues.