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. 2017 Dec 22;6(12):e006809. doi: 10.1161/JAHA.117.006809

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© 2017 The Authors. Published on behalf of the American Heart Association, Inc., by Wiley.

This is an open access article under the terms of the Creative Commons Attribution‐NonCommercial License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Study designs and treatment protocols. A, Stimulation of β‐ARs at the end of a 2‐hour reperfusion in the Langendorff model. B, Stimulation of β‐ARs 7 days after infarction in the working heart model. C, Cardiomyocytes were isolated from IPC or IPoC hearts and cultured for 24 hours. Cell shortening was determined before and after stimulation with ISO using a cell‐edge detection system. D, Perfusion of IPoC hearts 7 days after an infarction. After 10 minutes’ infusion with paroxetine, the CO was measured continuously for 5 minutes with additional stimulation using ISO. β‐AR indicates β‐adrenergic receptor; CO, cardiac output; IPC, ischemic preconditioning; IPoC, ischemic postconditioning; ISO, isoproterenol.