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. 2017 Oct 3;16(6):8930–8936. doi: 10.3892/mmr.2017.7694

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Serum levels of proinflammatory and anti-inflammatory markers in asymptomatic, TIA and stroke patients of <30, 30–90, 90–180 and >180 days measured by ELISA. (A and D) B1R levels of plaques in stroke patients of <30, 30–90, 90–180 and >180 days compared with asymptomatic or TIA patients. (B and E) IL-6 levels of plaques in stroke patients compared with asymptomatic or TIA patients. (C and F) IL-10 levels of plaques in stroke patients compared with asymptomatic or TIA patients. Compared with asymptomatic group, stroke patients showed increased B1R and IL-6 levels at <30 days and this increased serum B1R and IL-6 levels tended to decline after stroke from 30–90 to >180 days. By contrast, the anti- inflammatory marker IL-10 levels were gradually upregulated in stroke patients in spatiotemporal course from<30 days to >180 days. (*P<0.05, ***P<0.001 vs. asymptomatic or TIA group).