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. 2018 Jan 19;8:976. doi: 10.3389/fphar.2017.00976

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Copyright © 2018 Zhang, Wang, Zhu, Xu, Zhao, Zhao, Tang, Li, Zhou, Gao, Tian and Yao.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Carnosic acid-mediated protection against BDL-induced liver fibrosis involves the down-regulation of HMGB1, TLR4 and NF-κB. (A) The protein levels of HMGB1, TLR4 and nuclear NF-κB p65 in the liver were analyzed by western blot (n = 3). (B) Immunohistochemistry for HMGB1 in rat livers (x200). The data are presented as the means ± SD. ^∗∗P < 0.01 versus the sham group; ^##P < 0.01 versus the BDL group.