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. 2017 Oct 10;38(6):3327–3334. doi: 10.3892/or.2017.6019

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Copyright: © Meng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — BAF53a promotes proliferation, migration and invasion of glioma cells. The overexpression and knockdown interfered efficiency of BAF53a detected by real-time PCR (A) and western blotting (B), results showed shBAF53a-2 had the best knockdown effect and the overexpression effect was also satisfactory. (C) The MTT assays showed BAF53a overexpression promoted glioma cell proliferation, while BAF53a knockdown inhibited glioma cell proliferation. (D) The colony formation assays also showed BAF53a expression promoted glioma cell proliferation. (E) The Transwell assay showed BAF53a expression facilitated migration of glioma cells. (F) The wound healing assay showed BAF53a expression provided the faster wound healing capacities of glioma cells. (G) The Transwell-Matrigel chamber assay showed BAF53a expression facilitated invasion of glioma cells.