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. 2018 Jan 25;19:4. doi: 10.1186/s40360-018-0194-5

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Scheme showing the mechanism-based pharmacokinetic model for explaining increased fenofibrate absorption after food consumption. (V_stomach and V_duodenum: Volume of distribution in the stomach and duodenum, k_g (k_g’) and k_out: Gastric emptying rate constant and the elimination rate constant for calories, k_m&a: Metabolism and absorption rate constant of fenofibrate, k_el: Elimination rate constant, V_c/F: Volume of distribution, E_food, E_bile, E_Vc1 and E_Vc2: Parameters for explaining the effects of food on fenofibrate absorption)