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. 2017 Dec 22;10:269–276. doi: 10.1016/j.omtn.2017.12.011

Table 1.

Dysregulated lncRNAs in Neurodegenerative Diseases

lncRNAs Description Associated Disease Culprit or Bodyguard Biological Function
BACE1-AS transcribe from the antisense protein-coding BACE 1 gene AD culprit bind to BACE1 and increase the stability of its mRNA, thereby promoting the synthesis of BACE1 protein, and further increase the production of Aβ of cells
BC200 homologous with rodent BC1 lncRNA; the earliest specific example showed lncRNAs conservation AD culprit induce APP mRNA translation via association with FMRP and then aggregate the accumulation of Aβ in the brain
17A embedded in the human G-protein-coupled receptor 51 gene AD culprit impair GABAB signaling pathway by decreasing GABAB R2 transcription
NAT-Rad18 transcribed from the antisense of protein coding gene Rad18 AD culprit down the expression of DNA repair protein Rad18 and enhance susceptibility to neuronal apoptosis
51A an antisense transcript of intron 1 of the SORL1 gene AD culprit altering the spliced form of SORL1 mRNA and resulting in Aβ42 accumulation
GDNFOS transcribed from the opposite strand of GDNF gene AD culprit negatively regulate the expression of GDNF
HttAS_v1 antisense transcript of the Htt gene HD bodyguard reduce endogenous HTT transcript levels
BDNFOS antisense transcription product of BDNF HD bodyguard upregulates the transcription of BDNF and have a protective effect on neurons
NEAT1 a nuclear-enriched ncRNA essential for the formation and maintenance of paraspeckles HD bodyguard essential for the integrity of the nuclear paraspeckle substructure; increase viability under oxidative stress
HAR1F and HAR1R antisense transcripts of the first HAR1 gene HD bodyguard involved in neurotransmission, memory structure, and synaptic plasticity in the mature brain
DGCR5 a transcript of DiGeorge critical region 5 HD bodyguard include a genome binding site for REST and play an important transcriptional regulatory role in HD
MEG3 the human homolog of the mouse maternally expressed gene Gtl2, the first imprinted gene identified on the mouse distal chromosome 12 HD culprit alter gene expression in response to neuronal activity
ABHD11-AS1 homologous with rodent Abhd11os lncRNA HD bodyguard attenuate the toxicity of Htt mRNA
NaPINK1 transcribed from the antisense of PINK1 locus PD bodyguard stabilize PINK1 expression
AS Uchl1 antisense transcript of Uchl1 PD bodyguard regulate the expression of UCHL1 at the post-transcriptional level, thus promoting the translation process and increasing protein synthesis
HOTAIR antisense intergenic RNA transcribed from the HOXC locus PD culprit increase the stability of LRRK2 mRNA and upregulated its expression, thus inducing DA neuronal apoptosis
MALAT1 also known as NEAT2 and is a highly conserved ncRNA highly expressed in neurons PD culprit upregulate a-synuclein expression, thus driving the pathogenesis of PD