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. Author manuscript; available in PMC: 2019 Mar 1.
Published in final edited form as: J Steroid Biochem Mol Biol. 2017 Jul 29;177:149–154. doi: 10.1016/j.jsbmb.2017.07.005

Figure 1.

Figure 1

Western blots showing that phosphorylation of JNK1/2 and their downstream target C-JUN is increased by D2/CA in AraC-treated cells, and is required for the upregulation of BIM, and the DNA damage marker P-H2AX. The lack of change in P-p38 levels indicates the specificity of siJNK. CRK-L was used as an internal control for loading.