Skip to main content
. 2018 Jan 12;10(1):17. doi: 10.3390/cancers10010017

Figure 1.

Figure 1

Prevalence of potentially targetable mutations in PDAC. KRAS—KRAS proto-oncogene, GTPase [33,35]; TP53—tumour protein p53 [59]; SMAD4—SMAD Family Member 4 [19,67]; DDR—DNA damage repair pathway mutations [18,19]; CDKN2A—cyclin-dependent kinase inhibitor 2A [12,75]; RNF43—ring finger protein 43 [19,24]; BRAF B-Raf proto-oncogene, serine/threonine kinase [82]; MSI—microsatellite instability [84]. Dark colour signifies minimal range of mutation reported, while lighter shade signifies maximum range of mutation reported within different references.