Figure 6. The transient growth arrest caused by cell division inhibition allows a greater number of cells to resume growth.

By decreasing nusG expression, the oxidative stress‐induced OxyS sRNA leads to an increase in KilR. KilR prevents proper FtsZ assembly by sequestering the protein subunits. The inhibition of cell division allows for greater repair leading to a decrease in mutation rates concomitant with an increase in the rate of recovery.