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. Author manuscript; available in PMC: 2019 Feb 1.
Published in final edited form as: Mol Genet Genomics. 2017 Sep 11;293(1):119–128. doi: 10.1007/s00438-017-1365-6

Table 5.

Analysis of genotypic association between TFPI SNP rs10931292 and CAD under three different genetic models.

Population (n, case/control) Genetic model Before adjustment After adjustment

Pobsa OR (95%CI) Padjb OR (95%CI)
Overall CAD (4479/3628) Additive 0.089 1.06 (0.99–1.13) 0.088 1.06 (0.99–1.14)
Dominant 0.826 1.01 (0.92–1.11) 0.618 1.03 (0.93–1.13)
Recessive 0.001 1.25 (1.09–1.44) 0.005 1.23 (1.06–1.42)
Early-onset CAD (700/3628) Additive 0.944 1.00 (0.89–1.14) 0.644 1.04 (0.90–1.20)
Dominant 0.428 1.07 (0.91–1.26) 0.061 1.21 (0.99–1.46)
Recessive 0.167 1.19 (0.93–1.53) 0.049 1.35 (1.00–1.81)
Female CAD (1698/1329) Additive 0.524 1.04 (0.93–1.15) 0.634 1.03 (0.92–1.16)
Dominant 0.625 1.04 (0.89–1.21) 0.804 1.02 (0.87–1.20)
Recessive 0.038 1.27 (1.01–1.60) 0.169 1.19 (0.93–1.52)
Male CAD (2707/2242) Additive 0.058 1.09 (1.00–1.18) 0.079 1.08 (0.99–1.18)
Dominant 0.424 1.05 (0.93–1.18) 0.409 1.05 (0.93–1.19)
Recessive 0.006 1.28 (1.07–1.53) 0.016 1.25 (1.04–1.50)

Pobs, observed P value (X2 test); Padj, P value after adjustment for covariates (logistic regression analysis by adjusting for gender and age); OR, odds ratio; 95% CI, 95% confidence interval; Early-onset CAD, males age 50 years or younger; females age 55 years or younger at the first diagnosis of the disease; Additive model = GG/AG/AA; Dominant model = GG+AG/AA; Recessive model = GG/AG+AA.