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. Author manuscript; available in PMC: 2019 Feb 1.
Published in final edited form as: J Invest Dermatol. 2017 Sep 18;138(2):444–451. doi: 10.1016/j.jid.2017.09.004

Figure 4.

Figure 4

Epidermal deletion of integrin α9β1 promotes LNγ2 processing in vivo. Cryosections of 10-day, fully reepithelialized excisional wounds were prepared from control, α3eKO, α9eKO, or α3/α9eKO mice and stained by immunofluorescence with anti-LN-332 (to detect total LN-332) or anti-γ2L4m (to detect the L4 module of unprocessed LNγ2 chain). e, epidermis; wb, wound bed; arrowhead, BMZ. (a) Representative images are shown. (b) Unprocessed LNγ2 was quantified for each genotype as a percent of positive staining per field of wounded skin; scale bar = 100 µm; mean ± SEM.; n ≥ 4 mice per genotype; 1-way ANOVA followed by Newman-Keuls multiple comparison, *P<0.05.