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. 2018 Jan 4;15(3):3464–3471. doi: 10.3892/ol.2018.7728

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Copyright: © Xie et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — miR-96 promoted the expression of epithelial-to-mesenchymal transition associated proteins. (A) miR-96 was overexpressed in miR-96 mimic-treated MDA-MB-231 and MCF-7 breast cancer cell lines. ***P<0.001 vs. NC. (B) miR-96 was inhibited in miR-96 inhibitor-treated MDA-MB-231 and MCF-7 breast cancer cell lines. **P<0.01 vs. NC. (C and D) Expression of epithelial marker (E-cad) and mesenchymal markers (N-cad and vimentin) in miR-96 mimics and inhibitor-treated MDA-MB-231 and MCF-7 cell lines were determined by western blot analysis. NC, negative control; E-cad, epithelial cadherin; N-cad, neural cadherin; miR, microRNA.