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. 2017 Feb 3;4:19–30. doi: 10.1016/j.biopen.2017.01.002

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© 2017 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 4 — Hepatic gene expression of PPARγ (A), SREBP-1c (B), FAT/CD36 (C), ChREBP (D) and Plin2 (E) from the experimental groups in the post-treatment period. Endogenous control beta-actin was used to normalize the expression of the selected genes. Data are expressed as the mean and SD (n = 10 each group). P < 0.05 when: † compared to the C group; ‡ compared to the CM group; § compared to the F group (two-way ANOVA and the posthoc test of Holm–Sidak). Groups: C, control diet; F, fructose diet; M, Metformin. Abbreviations: peroxisome proliferator activator receptor gamma (PPARγ); sterol regulatory element-binding protein-1c (SREBP-1c); carbohydrate response element-binding protein (CHREBP); Fatty acid translocase (FAT/CD36) and perilipin-2 (Plin-2).