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. 2018 Jan 10;39(3):967–976. doi: 10.3892/or.2018.6204

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Copyright: © Xiong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Knockdown of MALAT1 partially reverses EMT of HT29 oxymatrine resistant cells. (A) MALAT1 was silenced in CRC cell lines by transfection of small interfering RNA. (B) Western blotting showed that transfection of siMALAT1-1 promoted E-cadherin while suppressed vimentin expression level in HT29 cells. (C) Knockdown of MALAT1 partially reversed the obtained migratory and invasive ability in HT29 oxymatrine resistant cells. (D) CCK-8 assay indicated that MALAT1 had no effect on proliferation of HT29 cells. Error bars represent median ± SD; *P<0.05; **P<0.01.