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. 2017 Dec 19;17(3):3599–3606. doi: 10.3892/mmr.2017.8310

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Copyright: © Hu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — LB reverses mitochondrial apoptosis. (A) Pretreatment for 3 h with LB restored the MMP disruption induced by 12 h L-Glu exposure. Scale bar, 100 µm. Data are expressed as the mean ± standard deviation (n=6). ^##P<0.01 vs. CTRL; *P<0.05 vs. L-Glu only. (B) Pretreatment with LB (3 h) reduced the levels of intracellular reactive oxygen species in DPC12 cells exposed to L-Glu (n=6). (C) Pretreatment with LB (3 h) inhibited intracellular Ca²⁺ overload (n=6) in DPC12 cells exposed to L-Glu. LB, Lycium barbarum; MMP, mitochondrial membrane potential; L-Glu, L-glutamic acid; CTRL, non-induced control; DPC12, differentiated PC12; PI, propidium iodide.