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. 2017 Dec 20;17(3):3681–3689. doi: 10.3892/mmr.2017.8327

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Copyright: © Huang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Luteolin inhibits pancreatitis-induced ADM formation. Hematoxylin and eosin staining of the pancreas in the (A) Ctrl, (B) AP and (C) luteolin groups on day 3. (D) Number of TC per field (magnification, ×200) in the Ctrl, AP and luteolin groups. Immunohistochemistry of CK19 in the (E) Ctrl, (F) AP and (G) luteolin groups on day 1. (H) Number of CK19(+) acini per field (×200) in the Ctrl, AP and luteolin groups on day 1. Immunohistochemistry of CK19 in the (I) Ctrl, (J) AP and (K) luteolin groups on day 3. (L) Number of CK19(+) acini per field (×200) in the Ctrl, AP and luteolin groups on day 3. *P<0.05. Scale bars, 100 µm. ADM, acinar-ductal metaplasia; Ctrl, control; AP, acute pancreatitis group; AP + luteolin, luteolin group; TC, tubular complex; NS, not significant.