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. 2017 Dec 18;29(3):208–213. doi: 10.1097/WNR.0000000000000948

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Copyright © 2018 The Author(s). Published by Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0/

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Fig. 2 — Effect of N-acetyl lysyltyrosylcysteine amide (KYC) on oxidative stress in NOD experimental autoimmune encephalomyelitis (EAE) mice. Frozen sections of spinal cords of NOD mice 70 days after EAE induction were immunostained with an antibody against myeloperoxidase (MPO) (a), chlorotyrosine (ClTyr) (c). The bar graphs (b, d) are the results of fluorescence intensity (FI) analysis of (a) and (c), respectively. Data are expressed as mean±SEM. One-way analysis of variance with Bonferroni’s test, **P<0.01, ***P<0.001, n=5/group.