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. 2017 Dec 11;7:1282. doi: 10.1038/s41398-017-0047-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a The correlations between the intrapair differences in childhood cortisol (x axis) and amygdala-modulated recovery (y axis) are shown. Note: Twin pairs selected for whole genome epigenetic analysis are circled in black. b–f The distribution of the twin differences for the full sample is shown for HPA activity (cortisol level; b; N = 299) and anxious behaviors (c–f; N = 184 to 215). The selected pairs are indicated by color showing their relative twin difference compared to the full sample (pair A = green; B = red; C = green)