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. 2018 Feb 8;10:17. doi: 10.1186/s13195-018-0344-4

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — Peak clusters showing significant interactions of B-cell chronic lymphocytic leukemia/lymphoma-2 (Bcl-2) AA and G carriers from the (a) default mode network anchored by entorhinal cortex (AA > G carriers); midline core subsystems with interactions of (b) AA < G carriers or (c) AA > G carriers; (d) executive control network by the dorsolateral prefrontal cortex (AA > G carriers). (x, y, z) = Montreal Neurological Institute coordinates. G carriers: blue; AA genotype: green. The colored lines represent the covariance strengths between seed and peak cluster with 95% CIs as boundaries. GM Gray matter, R right, L Left