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. 2018 Jan 4;52(3):787–803. doi: 10.3892/ijo.2018.4236

Table III.

Upstream regulators predicted to be either activated (in addition to the above top 5 upstream regulators), or inhibited in the IMR-32 compared to the SK-N-SH cells based on the statistically significant over- or underexpression of molecules within the IPA-derived dataset.

Predicted to be activated MYC, ESRRA, MYCN, INSR, HIST1H1T, HIST1H1A, HIF1A, EIF4E, EIF4G1, SREBF2, ANGPT2, FLI1, IL3, SREBF1, SCAP, RUVBL1, SP1, CD40LG, EPAS1, KITLG, ADORA2A, PGR, KAT5, PSEN1
Predicted to be inhibited ALYREF, ANK2, ATG7, CCDC88A, CHTOP, CPSF1, CPSF2, CPSF3, CSTF1, DDX39A, DDX39B, DENR