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. Author manuscript; available in PMC: 2018 Feb 12.
Published in final edited form as: J Endocrinol. 2017 Aug 14;235(2):R63–R76. doi: 10.1530/JOE-17-0076

Figure 1. A schematic representation of fetal pancreatic and β-cell dysfunction in models of IUGR and potential mechanisms of glucose regulation for β-cell dysfunction.

Figure 1

The fetal pancreas response to glucose is represented by the gray lines. Pancreatic dysfunctions associated with models of IUGR are depicted by red lines. A fetal sheep islet is at 90% of gestation is depicted in the micrograph and has been immunostained for insulin (β-cell; blue), glucagon+somatostatin+pancreatic polypeptide (red), and vasculature (GS1; green).