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. 2017 Nov 6;9(12):741–754. doi: 10.1177/1758834017737471

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© The Author(s), 2017

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 6. — DNM3, p65 and p53 are the potential clinical diagnostic markers. In summary, DNM3, p65 and p53 had the similar trend in brain and blood exosome both for original (a) and recurrent GBM (b). (c) DNM3, p65 and p53 and internal control β-actin protein expression of brain and blood exosome both for original and recurrent GBM were analyzed by Western blotting. Relative expression values represent mean and SD from three independent experiments. Quantitation by densitometry was shown on below.

GBM, glioblastoma multiforme; PCR, polymerase chain reaction; SD, standard deviation.