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. 2018 Feb 12;8:2846. doi: 10.1038/s41598-018-21270-8

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Ang II infusion does not affect oxidative stress in adipose tissue, adipokines, and adipocyte differentiation in WT and TG mice. (a) Relative NOX2, p22^phox, p47^phox, and p40^phox mRNA expression levels in WAT of WT and TG mice after 2 weeks of vehicle or Ang II infusion. Values are expressed as mean ± SE (n = 5 in each group). (b) Relative tumour necrosis factor-α, interleukin-6, and adiponectin mRNA expression levels in WAT of WT and TG mice after 2 weeks of vehicle or Ang II infusion. Values are expressed as mean ± SE (n = 5 in each group). (c) Relative PPARγ and C/EBPα mRNA expression levels in WAT of WT and TG mice after 2 weeks of vehicle or Ang II infusion. Values are expressed as mean ± SE (n = 5 in each group). WT, wild-type; TG, ATRAP transgenic mice; Ang II, angiotensin II.