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. 2018 Feb 12;8:2797. doi: 10.1038/s41598-018-21143-0

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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recMgIFN-γ limits replication of vesicular stomatitis virus in BVK168 cells. Cells infected with the VSV*ΔG(FLuc) strain expressing firefly luciferase were treated with either IFN-αB/D (5 ng/ml) as a known positive effector; with the negative control protein LEA; two concentrations of recMgIFN-γ (1 and 0.2 ng/ml), or left untreated as control (ctrl). The bioluminescence signal represents viral replication which is reported as percentage relative to untreated cells. Shown are mean ± SEM (n = 3 experiments, each with 2 replicates). Statistical analysis: One-way Anova (F(4, 10) = 37,24, p < 0.0001) followed by Sidak’s multiple comparisons test, **p = 0.0003, ***p < 0.0001.