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. 2018 Feb 12;18:174. doi: 10.1186/s12885-018-4021-6

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Expression profiles of the AKAP gene family in three different cohorts, tumors ordered by PAM50 subtype, genes are orderad by hierarchial clustering. a TCGA RNAseq expression profiles from primary tumors (n = 522). Genes are ordered by hierarchical clustering p < 0.0001 by ANOVA; q < 0.000072. Intrinsic subtype calls for the TCGA data were taken from the original manuscript [1]. b Risk Cohort microarray expression profiles from primary breast cancer tumors (n = 182). c Cohort 1 microarray expression profiles from metastatic lesions (n = 120)