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. 2018 Jan 26;9:83. doi: 10.3389/fimmu.2018.00083

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Copyright © 2018 Xiong, Qiu, Li, Qiu, Guo, Liu, Wan, Li, Wu, Wang, Zhou, Dong, Du, Chen and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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MicroRNA-374a (miR-374a)-mediated fortunellin-induced modulation on phosphatase and tensin homolog (PTEN) expression. The cells were incubated without (control) or with fortunellin for 24 h. (A,B) miR-374a could bind to the 3′-untranslated regions (UTRs) of PTEN mRNA and repress PTEN expression in Caco-2 cells. (C) miR-374a inhibitor inhibited fortunellin-induced modulations on MLCK expression. The data are presented as the mean ± SD (n = 3). **P < 0.01 vs. the miR-374a negative control group, ^##P < 0.01 vs. miR-374a or PTEN before fortunellin treatment, respectively.