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. 2018 Feb 14;15:8. doi: 10.1186/s12979-017-0108-1

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — NLRP3 and redox alterations in the pathophysiology of Alzheimer disease: Redox equilibrium is maintained by the balance of ROS and antioxidants. If ROS prevail, cells undergo oxidative stress and NLRP3 inflammasome activation. If antioxidants prevail, reductive stress may occur with NLRP3 inflammasome suppression