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. 2018 Feb 20;4:4. doi: 10.1038/s41537-018-0044-x

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Venn diagram showing the number of genes differentially expressed in Brodmann’s areas (BA) 9 (n = 60), 10 (n = 560) and 33 (n = 35) from subjects with schizophrenia compared to controls. The level of expression of no gene was significantly different in all three cortical regions from subjects with schizophrenia. Levels of expression of a limited number of genes were changed in more than one cortical region (BA 9 and BA 10 = 3, BA 9 and BA 33 = 2, BA 10 and BA 33 = 3)