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. 2018 Jan 4;46(4):1895–1911. doi: 10.1093/nar/gkx1306

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© The Author(s) 2018. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited. For commercial re-use, please contact journals.permissions@oup.com

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Figure 4. — Epigenome landscapes of ARV-PBS, ARFL-PBS and Common-BS. (A) Heatmaps showing normalized read intensities for histone modification ChIP-seq and histone variant H2A.Z ChIP-seq at ARFL-PBS, Common-BS and ARV-PBS. Magenta and cyan indicate high and low read intensities, respectively. (B–E) H3K27ac, H3K4me2, H3K4me1 and histone variant H2A.Z ChIP-seq signal profiles for ARFL-PBS (red), Common-BS (purple) and ARV-PBS (blue) in 22Rv1 cells without (left) or with (right) R1881 (methyltrienolone) treatment. Zoom-in views of the y-axis for common-BS and ARV-PBS were displayed on the top-left corner of the panel (E).