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Schematic of platelet’s injury site-selective adhesion mechanisms (platelet GPIbα of the GPIb-IX-V complex binding to vWF, and GPIa-IIa as well as GPVI binding to collagen) and aggregation mechanism (fibrinogen-mediated bridging of active platelet surface integrin GPIIb-IIIa), and various hemostatic technologies inspired by these mechanisms.
