Table 2.
Subpopulationa | N | H | No. of monomorphic SNP markers | H S | r̅ d | P (r̅ d) | MAT1‐1 % (P χ2) | HfMV1 % (no. tested) |
---|---|---|---|---|---|---|---|---|
EPW‐E | 19 | 2.94 | 3 | 0.354 | −0.0052 | 0.97 | 32 (0.3) | 67 (15) |
LWD‐E | 13 | 2.56 | 6 | 0.309 | 0.0130 | 0.14 | 54 (0.8) | 20 (10) |
PND‐M | 14 | 2.64 | 0 | 0.451 | 0.0523 | <0.001 | 71 (0.3) | 100 (13) |
BWY‐P | 10 | 2.30 | 3 | 0.365 | 0.0098 | 0.14 | 80 (0.2) | 46 (13) |
ISC‐P | 14 | 2.64 | 7 | 0.343 | 0.0311 | 0.14 | 36 (0.5) | 87 (15) |
PTW‐P | 20 | 3.00 | 5 | 0.294 | 0.0107 | 0.64 | 40 (0.5) | 67 (24) |
Total | 90 | 4.50 | 0.411 | 0.0206 | <0.001 | 49 (0.9) | 67; P = 0.0007 |
28 SNP markers were tested on each population.
N, sample size; H, Shannon–Wiener index; H S, Nei's gene diversity (expected heterozygosity); r̅ d, standardized index of multilocus linkage disequilibrium (Agapow & Burt, 2001); P, value for significance of r̅ d. The MAT1‐1 allele frequencies at each site were tested for deviation from a 1:1 ratio using a χ2 test with 1 degree of freedom. Homogeneity of the frequency of HfMV1 across sites was tested by a χ2 test (5 d.f.).
For details of subpopulations see Table S1.