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. 2018 Feb 28;9(3):328. doi: 10.1038/s41419-017-0025-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Transmission electron microscopy of spinal cord neurons from 3-month-old β-Gal^−/− and β-Gal^+/+ mice shows evidence in the affected mouse of an expanded lysosomal compartment with enlarged lysosomes filled with membranous material due to accumulation of GM1-ganglioside. Scale bars: 1 μm; lover right panel 0.5 μm. Adapted from the original article Tessitore et al., 2004¹⁰⁸ with the permission of Elsevier